RT Book, Section
A1 Al-Mourgi, Majed A.
A1 Bueno, Raphael
A2 Sugarbaker, David J.
A2 Bueno, Raphael
A2 Colson, Yolonda L.
A2 Jaklitsch, Michael T.
A2 Krasna, Mark J.
A2 Mentzer, Steven J.
A2 Williams, Marcia
A2 Adams, Ann
SR Print(0)
ID 1105840416
T1 Techniques for Dilation of Benign Esophageal Stricture
T2 Adult Chest Surgery, 2e
YR 2015
FD 2015
PB McGraw-Hill Education
PP New York, NY
SN 978-0-07-178189-3
LK accesssurgery.mhmedical.com/content.aspx?aid=1105840416
RD 2024/04/20
AB Benign  strictures  of  the  esophagus  usually  result  from  scarring  and  subsequent  tissue  contraction  secondary  to  esophageal  wall  injury.  This  pathology  is  caused  in  most  cases  by  long-standing  gastroesophageal  reflux  disease  (GERD),  often  in  association  with  one  of  the  esophageal  motility  disorders  (e.g.,  achalasia,  diffuse  esophageal  spasm,  or  aperistalsis).1,2  Endoscopic  dilation  of  benign  esophageal  strictures  that  are  refractory  to  medical  management  is  a  less  morbid  alternative  to  surgery.  Approximately  20%  to  30%  of  cases  are  unrelated  to  GERD,  and  their  treatment  usually  is  more  challenging.  Examples  include  strictures  arising  from  complications  of  surgical  anastomosis,3  injuries  caused  by  caustic  ingestions,  early  and  late  consequences  of  external-beam  radiation,  esophageal  sclerotherapy,  laser  or  photodynamic  therapy,  medication-  or  pill-induced  esophagitis  that  is  associated  with  numerous  medications  (e.g.,  alendronate,  ferrous  sulfate,  nonsteroidal  anti-inflammatory  drugs,  phenytoin,  potassium  chloride,  quinicline,  tetracycline,  and  ascorbic  acid)  but  most  often  aspirin,  and  rare  dermatologic  diseases,  including  epidermolysis  bullosa  dystrophica,  among  others.